mmu-mir-126: Conditional ablation of Dicer in hematopoietic stem cells (HSCs) has shown that mmu-mir-126, along with other microRNAs such as mmu-miR-29a, mmu-miR-130a, mmu-miR-155, and mmu-miR-125a/b, plays a role in controlling HSC differentiation by targeting different genes [PMC6829453]. Specifically, mmu-miR-125a has been found to increase stem cell quantities by targeting BAK1 [PMC6829453]. In a study on offspring from OVA-immunized mothers, it was observed that the downregulation of mmu-mir-126 was associated with reduced IL-17-producing γδT cells [PMC8234718]. This suggests that maternal OVA immunization can influence the thymic expression of several microRNAs including mmu-mir-126 [PMC8234718]. In another study on mouse aortic endothelial cells (MAECs), miR-126 levels were modulated using miR-126 mimics and inhibitors [PMC5855744]. It is worth noting that inadvertent deletion of mmu-mir-126 has led to misattributed phenotypes in previous studies on angiogenesis defects [PMC3675212]. These findings highlight the importance of understanding the role of mmu-mir-126 in various biological processes and its potential implications in disease development and treatment.