rno-mir-132: Rno-mir-132 is a microRNA that has been found to target matrix metalloproteinases (MMP) 9 and SPRY1, suggesting its involvement in cardiac fibrosis and hypertension [PMC5413218]. Treatment with angiotensin II (Ang II) for 24 hours has been shown to induce cardiac fibrosis and hypertension, with rno-mir-132 playing a role in this process [PMC5413218]. Rno-mir-132 is one of the differentially expressed miRNAs in the AKI model group compared to the control group, and its expression is reversed following MSC treatment [PMC6625446]. Rno-mir-132 is also one of the common miRNAs that are upregulated in the AKI group and downregulated in the MSC treatment group [PMC6625446]. In healthy Wistar islets, rno-mir-132 responds to glucose stimulation at different concentrations [PMC3072418]. The expression of rno-mir-132 shows fluctuations over time, with downregulation at certain time points followed by a return to baseline or further reduction [PMC5979605]. Rno-mir-132 has also been found to be downregulated in DHT-treated rats, suggesting its association with promoted thecal hyperandrogenesis [PMC3682887]. Hyperglycemia can upregulate the expression of rno-mir-132, but prolonged exposure may lead to a resetting of its levels back to control levels [PMC4620849]. The full sequence of zlg-mir-132 shows similarities with other species' miR-132 sequences such as hsa-miR-132 and tgu-miR-132 [PMC4636775]. In PC12 cells, modulation of rno-miR-132 expression can be achieved through antisense or mimics transfection methods for further study purposes [PMC3420912].