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Homo sapiens (human) long intergenic non-protein coding RNA 2672 URS0000EEE793_9606

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LINC02672: LINC02672 is a long non-coding RNA (lncRNA) that has been found to be significantly up-regulated in various cancer cell lines and cancer tissues compared to normal cells and tissues [PMC8093983]. It has been shown to be correlated with the expression of IDO1, NRP1, and TNFSF4 in pan-cancers [PMC8093983]. Additionally, LINC02672 is associated with the abundance of CD56 bright natural killer cells, mast cells, and immature dendritic cells in pan-cancers [PMC8093983]. It is also significantly correlated with predicted SNV-derived neoantigens in bladder cancer and uterine corpus endometrial carcinoma [PMC8093983]. Another lncRNA, PSORS1C3, has been found to be significantly correlated with the expression of CD160, CD40, and TNFRSF14 in pan-cancers [PMC8093983]. PSORS1C3 is also associated with the abundance of activated CD4 cells and gamma delta T cells in pan-cancers [PMC8093983]. RP11-89 is another lncRNA that is highly expressed in tumor samples along with PSORS1C3 and LINC02672 [PMC8093983]. On the other hand, MIR100HG is highly expressed in normal samples [PMC8093983]. These four lncRNAs (RP11-89, PSORS1C3, LINC02672, MIR100HG) were used to construct an immune-related prognostic lncRNA signature (IRPLS) that could potentially serve as a target for individualized immunotherapy for bladder cancer patients [PMC9604712]. References: - PMC8093983 - PMC9604712

Genome locations

Gene Ontology annotations

Sequence

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AGUCCAUGUCUCUUUCACUCACUGUGGUCAGCUUCCACACCAUUUCUUUGGUGUGGCUUGGCAAGAACCUCAGGUGUUACAUCUUGGCGAGCCAGACAGGAGACUCCAGAAAAGGUAUCUAGAUCAUCAUGCAGGGUGAUUUUCCUGUACCAGUCCAAUGCCUCCAGAGGAAGAUCAUACAUUUGCCAUUUUACUGCUUAGUACGCAUGCUUGAGCCUGCUCGCCCAACUGCUGAGAUCUUAUUCAGAAACUGCUGAUCACCAACUCCAGGUGUUUUUAUUACACAAGACAAAAACGGAAAAACAGUAACAAAUGAUUGGUUAAGCCUUUCAAAACAAAGAAGUUAUGCCUGGAGUGGGAGGAACAUUACACUAUACUUCUUUCUCCUCCUUCAUUAGUGAAGGUCACUGGAAUAGGUUCAUUACACUCAAGCAAAGGCCUGGGGAAAUGAUAGAAUUACCUCUGUUGACCCAGAAGAGCACCAAAAGCACCAGUGUGCAGAAAUCAGAGACCUCAAGCUAAAAGUCAUAAAAGGACAAGAACCUGGAAGUCGUCGAACUGUGGAAGCAAAAGACCUGUAAUGCUCCUGCUUGCCGAGCUGUGAGCAGUGGUAGUAAAAGAGCAGUAACAUUCUCUCCUGCUUGCCAAACAGGAGAGAAGCCACUGGCACCACUCCCUCCCACUCACUGAACUAUAGAAGAGAAAAAAUUGAAACACCAAAAUUGGGGAAAUGCGACUUUUGCGAAAAUUUGAAAGUGCAUGCUUCACAGAAGGAAGGGGGAGCUGGUCUAUUAUGUCUCACUAUAACAAGACUAAAAUUUUGCAAAUAAAUUGUUCCUGCCAUAAUUUAUCCUUGGUAGAAAUGAAGAAAUUGGAGAGAGAAAAAAUUGGAUUCUAGGUCCUGACCAUUGUUUUUGAAUUUUUAUUAUUUUCCUACAAUUUAGGCUAAAUUCUGAACUAUUUGCUGGCUACAAGAGGCCUCUAAAAAAGAGCUGGGUUUUAGUUUUCUUCAUGCUCUUUAAAAACUGGCAUAUAAUGGUGUGUAUCUCUCGUUGUUUUACUUCUAAGAAACUGAAGGCAUGAUAUUCUGAAGACUAGAGGUGAUUCAAUAGCCUAUGAAUCUUCUUCAUUUGGAACCCCACUGGGCCUGAUCUGUUUCCCAUCACCAAUGCACUUCUGCUAAACCUAUAUCCUAUCAAGUACUCUCCCUAAAUGCUCAGAGACCAUAACAGAAGAUGAGGGUGUGUGAGAUUAUAAAAGCCAAAUUAGUGGAGUGGAAUCUGGAGGCUAAAGCAACUCCAUCUUGGAUGCUAAUCUGCCAUGUUGGCUUCUGAUUAACCCCAGUUCUGGAGGGCCUCUGAGGUUUCCAAUUUAUCUACUGUCCCUUGUGUAAGAACAAGUACUUAUCAUAAAUCCUGCUAUUAGGGCAAAUUUCUACCCAUUCCCUCUGAAGCAUGUGCACCUCUCCUCUACAUGUAAUCUCCCCUACAAUAGAUAAACCCCUCUGGGUCUGGGGGCUAAUAGUGUGGGAACCCAUCAUCUUGCCUCACUGCUGCACAAGCCACAGGUGUGGCUUCUGUUUCUAAGUCCCUAUUAAAUGUUUCUAGAUACCA

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Publications