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Homo sapiens (human) ZMIZ1 antisense RNA 1 (ZMIZ1-AS1) URS000075ADF9_9606

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ZMIZ1-AS1: ZMIZ1-AS1 is an antisense noncoding gene that shares a bidirectional promoter with ZMIZ1 [PMC8259458]. It is a long noncoding RNA (lncRNA) that has been found to be highly expressed in glioblastoma (GBM) and is associated with poor prognosis [PMC6874448]. ZMIZ1-AS1 has been shown to regulate several GBM-related genes, including CPEB4 and RNF43 [PMC6874448]. It is also one of the genes with the highest proportion of CpGs designated as "high 5hmC" [PMC8451154]. The function of ZMIZ1-AS1 is still unknown, but it has been associated with colorectal cancer and patients' survival in the Korean population [PMC8394133]. In GBM, ZMIZ1-AS1 forms a separate independent network [PMC7922682]. In addition, it has been identified as one of the lncRNAs significantly associated with overall survival (OS) in GBM patients according to random survival forest analysis [PMC6302404]. Overall, ZMIZ1-AS1 appears to play a role in GBM progression and prognosis, but further research is needed to fully understand its function and potential as a therapeutic target.

Genome locations

Gene Ontology annotations

Sequence

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ACGUUUUGCAGGCUGCCACCGUGUCGGAGGCGAGGCGAGGAAGGGAGCUGGAAUAACAAAGGUGGCAGCUGAGCAUCCCUGGAGAGGGUGGGUGGUAUGAAAGCACUUCCAGACCUCUAGGGACACCAGGGAGUCAUGGUCCCAGCACAUUGCUGUGUGAUUGAGCCCCUCCUCAGCCUGUGGGUGGCCUAAGUUCACAGGGAGGUAAUGGGGUAGAUUGGAUACCUCUGGGGUCUUGGAAGAAGCUAUGACUUAUUUACUGUCUACUAUGUGAGUUUUGGGCAUCCUUCUAUCAGCCCUUAUCCCAGGGCAGGAAGAACAGCCUUGAUCCUGGAUCCCAGGCACAGCUGGGUGUCAGACCUGGAGGAGAAGCUCCCAGAGUGACCAAGAUAGAAUCGGCAUGCAGCUGAAGCCACUUGCUCAGAGCCGCACAGCAAGCCCACCACCCUGAACCUCCAGAUUACCAACUCAGCAUUGUGUCCUGACAGUGUCAUCAUAGAUGGUCAACACCGUCACCUGGGCAGCACUGAAGCCCAGAGCGGGAGAGACUGGCUCAAGACCAUGGGCCCUGACAUUCUCCAGCUUUCAUGGUGUUCCUGCAAUCCAAGUGCCGCUUAUCUCUCUACAGGUCCACCAGAUGACCGGCUGGCCUCCUAAUUGGGCUUCCUGCAUUCCUGCCUGUGCUCGGAGCUCCCUUCUCCACUUGCCUGCUGGAGGAGGUUUACAAACAAAAUCAGAUCAUGGCACUCCCCUGGGGCCCUGCAUCCUGUGGCCAGCAGGUCUUGGAAACGCCCCUGUACCCCAGAAACUUCCUGAUCAGUCCCUGGUCAGUGUAUAUCAUGCUUCCUGUGACAUGACAUCAACUUUUCAGUGACUUCCACUGGUUUUGGCCCAGCUCGAAGAGCCUGACACCUCUGCAGGGAAUUCUACCAGGGAGCUCAGGAGGGGGAAUUGCACUGAAGAUUUGUCCCAUCGUGAAGCAAGAGCACUGGAACUUACACUCCACCAUCAGGCCCUGUCACAGGAGAACAAAGAAGGAAGGCAGAGGAGAUCAUGCUCCGGCCAGCAGGGAAUCUCCAUUUUUUUCAGCCUCAUACCUUGGAAAGUACAAAGGAGUGAGAGCUGGGACUACCAGCCAGAGGGUUCAUGGAGGUAGUGGGAGGGGAAGAUGGGUUCUGCAUGGAGCCACUCCAGGGACUUUUCUUCUGUCUCACAGCCUGACAAUCACCUCUAGCUGUUCUCAGUCCCAUUCUCAUCAAUGACCUCGUCUCCUGAUUUUGUACAAUAUCUAGGUGACCUUCUCUUCUUUCCAUCUUCAAACGCUUGAUCAUUGAUGCUCCCUUUUCUCUUCUCUCUCCUCCUGGGAAAAGAGAAGGACCAACAUCCCCCUUUCCCUUCCUCCUGUGCUGUCCCGACCCCUCUGAGACCUGGCUCUAGCAGUAAGUCCCUCUCUCUUCUGCAGCACCAGCCUCUGCCCAUCCCUGCCCUCUGCACACAAAGCUCUCACAUUGUCUGCACUUUAAGAACUUUUGUGGCAAACCCUGGCAAAUCCUGCCACUUGUUUCCACUCUUCUCAAAUGGCUUUGAGGUCAUCAUUGACCUAUCCCAUUUGACCCUGUCCUGUAUCUGAUGUUGUACCCUUUCUCCUUGAUCCCACUCUGGCAACUCUCCCACUCCCCUUUCUUGGAUCAACUGUCUUGCUCCUCAGUCUUGAUCAAUGCCUCCUUCUCUCUCUUUAGCCUUGGCCCUUUUUGGCCAGGGAAAUAAAUGUGAAAUACAUAAAGCUUCAUUUUAUCUUAUGAUUAUAGCUAUGUUGAUGUUACCCAAGACCUAAAAGUCACACUCUCUAAUUACACUUCCCUCCUGUGUUCUGGGUCCUCUUGGACCAGUCCUUACUCUGGCUCCCCUCCUGGAUGUCUCCCAGACUUCUAAUUAGAAUUACUGUUUCCUAUAUCAAGCUAAUUAUCUUUCCCACCAACCUCCUCCAUCCUUGGGUGAAAGCAUCACUUGGUUGCAAAAGUCAGAAAUCUGGGUUUCAUUCUUGCGGUAGGUAGAAUAAUGCCCCCAGGCCCCAGGUUUUGACAUCCGAAUCCCUGGGACUUUGCAGAUGUGAUUCAGUUUAGGAUUUUGAGAUGGGGAGAUUAUCUGGGAGAGCCUGAUGUCAUCAUAAGGUUCUUAUAAGAGGGAGGCAGGAGGGUUAGAGUGAGUAGUAAGAGAUGCAACAGUGGAAGCAAGAGGUUGGGGUGAUGUGGCCACAAGCCCAGGAGUGCUGACAGACAUCAGAAGCUGGAAGGGACAAGGAAUGGUUUCUCCUCUGGAGCCUCCAGAAAGAACCAGCCCUGCUGACACCUUGAUUUUAGCCUUGGAAGACUCAUUUUGGACUUCUGACCUUGAACGUUGUAAGAGAAUAAAUUUACAUAUUUUAAACUGGUAAAAAAAAAAAAAAAAAA

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Publications