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Homo sapiens (human) SMAD5 antisense RNA 1 (SMAD5-AS1) URS0000759D45_9606

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SMAD5-AS1: SMAD5-AS1 is a gene that has been studied in relation to various diseases. However, there is limited data available on its association with nasopharyngeal carcinoma (NPC) [PMC6923203]. In a study on diffuse large B-cell lymphoma (DLBCL), it was found that SMAD5-AS1 was downregulated. Additionally, upregulation of SMAD5-AS1 was found to suppress cell proliferation by inhibiting miR-135-5p and upregulating APC expression [PMC7693482]. The study used glyceraldehyde 3-phosphate dehydrogenase (GAPDH) as an endogenous control for SMAD5-AS1 and APC, and U6 for miR-135b-5p [PMC6420660]. In summary, SMAD5-AS1 is a gene that has been studied in relation to various diseases. However, there is limited data available on its association with nasopharyngeal carcinoma (NPC) [PMC6923203]. In diffuse large B-cell lymphoma (DLBCL), downregulation of SMAD5-AS1 was observed. Furthermore, upregulation of SMAD5-AS1 was found to suppress cell proliferation by inhibiting miR-135-5p and upregulating APC expression [PMC7693482]. The study used glyceraldehyde 3-phosphate dehydrogenase (GAPDH) as an endogenous control for SMAD5-AS1 and APC, and U6 for miR-135b-5p [PMC6420660].

Genome locations

Gene Ontology annotations

Sequence

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UGAUCUGAAAGGAAUGGAAGCACAAAAUGAUGAAUAAGGUAUUUUUAACAAAGAUACAUGGGUAAAUUAACAGCAGUAAUGUAAAAAAGACUGAGGGAGCAACAAUGUGGAAAGGGAAGGAAAGGAAGCUGUAUAGGAACAAUCACACAAAUAAUCAUCAAAAUUAAACUUUAUAAAUCUAAACAUAGUAUCUACAACCUUUAUGAAGGUUAAGUUUUGUUAAAUUGUCUAAGUUUUGCAUUUUUACAAUUUUCACUGUAGAGAAAAAAAUAUUAAUAGUUACCAAAGGAAUAAAAGACUAAAAUAAUUUGUUAUACAUCUGCAAAUAGCUCCUUAAAUUUGGAACACUAAUUUCAUAAUGAUAUUUGGAUCCAUAAAUGUGAAACUGUCACACUGAAUGUAGGUACUGUUUUCCCCACAAUGGAUACUACUGCAUUGGCAUCUAGCAGAAAUUGUAAGCUAUUUGGAAUGUUACUAAACCUACUGGGACUAAGGUUUUCAAUCUUUUGUCUUUCAAAUUAAAAUUAAAAACUAAUUCACUCUGGGCAAUUCAGGUACUCUAAGGAAAUUCAAGUACUCUAGUUUUAAAUACGAAUUUUCAAAAACGCAACCCAAGUACCCUUUCCUCUCAACUUCUGUGAGGCCAAGCCACGACCUCCACCUAUUCAAUCUUUGCUGCUAGAUAACAACCCUUUUGGUAGUGCUAGGCAACAGGCAUGAUGUGACAAACCUAGACAUCAAAACUUUUCCACAGAAAGAUAAGGUAAACAAGAUAUACUUAGCGCCACACCCAUUUAAAUGCACAAGCAACCAAAAUUGCUCCCUCCCCCCGCAACACCCCCCCCCCCCCCCCAAAGCUCCAGCUGGUCAGGAAACAUUGUCUUUACAAUCAAGAUAAACAUCUGGCUCAGGGUGUUCAGCCAUUCCUCACCUACAGGCUUGCCUAAGCCCCAUUCUCCAAUGCCAUCACCACCAGAACCGGAACAUUCUGUUGGGAAGCCGGCAAACGUCCAAAUACCCCAAGUAUCCUCGCCCGAGUUCUGCAACCAAAAAUCUGUUUUGGCUACUGAGCAUGCUCAGACUUAAGACGGGCGCGCCACAUGGGAGUUUUCAAAGCUUAGGCUCCAAACUCCACGUAGUAACAGCAGCUGAAAGGAGGAGAGGGGAAAAGGGGAGGGGAAGAGACUGGAAUAUUAAUUUGAGCCCUGAUAUUGAAGUAUAGGACCCGGCCAGCGGCUGAAUAUGGUGAAGGCAAAAAGACGCUGCUUUGGCAUUCUCUUUGCAGCACAGAGAAGUUUACAUGAAGUAGAAGAAAUCCUUGCAGUGGUGAAAACUGACAUGGCAAGUGGCAGAAACCAGGUCUAACUACCAGCUGAUUGCAGUAGCCGGUCACAACUGCCCAGAGUGUAAUUCCCACCGAUAAGCGCGGAUCCUUAUCUUUGAAACACAAAGCGACGGCGGCACGGAUUCAUCAAGCCAUUGGACAGCUUCGCAGUGCAGCAGCCCAGGCUGCCCGGCCGAGCACCGCAGUCCUAUCAAGGUUUGCUGUAGCUCAGAAGGCUCUGCCUACAUGACUACUGCAUACCCUACUGGGAGAAUUGGAAGAAUGUGAUGAUGGUAGGAGUCACCCAGCAUAGAAGCAUGAGUGAAUGACAUCCCAGAGAUCUGCAAAGGAUCCUGGUAGCCACUUCAAAGACUUUUUCUCCCUUCCUCAGACAAUCAUGGCACAUGGACAUCAGAGGAAGAAAUGGCGAAGUUUGUAUGACUCCUCCAGCAGAGUCCUUGGAAACAAUAAAGCUUCCUUUCCAAAAAAAAAAAAAAAAAAAA

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Publications