v24

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Homo sapiens (human) small nucleolar RNA host gene 10 (SNHG10) URS000075DE81_9606

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SNHG10: SNHG10 is a long non-coding RNA (lncRNA) that upregulates the expression of INHBC, activating the TGF-β signaling pathway and suppressing NK cytotoxicity [PMC8507338]. In glioma, SNHG10 is involved in a competing endogenous RNA (ceRNA) network [PMC7725120]. However, the role of SNHG10 in Parkinson's disease is still unknown [PMC8805890]. In osteosarcoma, SNHG10 promotes glucose uptake by interacting with miRNAs and upregulating TRIM66 through miR-1271-5p sponging [PMC9663463]. SNHG10 is positively correlated with various other lncRNAs, except for specific cases [PMC8589390]. SNHG10 overexpression increases the luciferase activity of the wild-type FZD3 reporter, which is abolished by miR-182-5p [PMC7674123]. SNHG10 is upregulated in osteosarcoma and promotes the expression of BIN1 in vivo [PMC7448318] [PMC8837780]. In hepatocellular carcinoma, SNHG10 upregulates c-Myb expression and promotes cancer cell proliferation, migration, invasion, and epithelial-mesenchymal transition (EMT) [PMC8837780]. SNHG10 expression is higher in cancer samples compared to matched adjacent normal samples [PMC8523833], while its levels are decreased in renal cell carcinoma cells along with other lncRNAs such as SNHG9 and DANCR [PMC8100577]. Overexpression of both SNHG10 and SNHG12 may lead to worse relapse-free survival in bladder cancer patients [PMC8100577].

Genome locations

Gene Ontology annotations

Sequence

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AGCCUCAUCCUACUGCCUUACUAUUGGUCGUCGGCAACCGCUUUGUUAGUUAAUUGGAGAGACUGGAUGUCCAUCAGGGUCACGCUUGCAGGGCAACGAGAGGCAAAACAAGAGGGAAGACGACUUUCCUCCUGUGAGCCAAUGAGGCCAGCUGGACUACGCCGAGACAACUGGGAGAGGCGCGGGACUCGCCCGUUCCGCGGAACGCCGGGAAGGGGUCACCUCCUGAUGAAGUUUCCGGUUCCGGUGUCAGCGGCGGUUGAAUUGCCAUGGCAAUGCGGUGGGCGCGCGCUUGUCGUGUUGGUCUCUUGGGAGGUAGCGGGGCUAGGCCGGGCGGGUAUCCGCCUCUCCCAGCUUAGGUGAGCGUCCCCGGGCGCCUCCGGAGCGCCGCGGCCGCAUGCAGUUCGUCGUGGCGGGGAGCCGGAGCCUGACCGGGGUUCCAGCGCUCGGGCCGUAGCCUUGGCUCCUGGACUUUCCCUGGCUCCGCCGCCACGUGGGAGCUGAGGCUCUGGGGCUUCCGCCUCCGGCGCGCGAUUAUUUCUCUAGAACAGUUUUCAUUUUUAAAAUUUGUAAAGCGCUUUUGCCUGUGUGAUUUCCUCUGGGUUUUUUUUUUUUUUCUUCCUUUUUGUAGAGACGGAAUUGGCGGCGGGGGCGGGGGGUCGAUGUCUCACUUUUUUGCCCAGGCUGGUCUCGAACUCCUGGCUUCAAGGGAUCCUCCUGCCUCGGCCUCUUAAAGUGCUGGGAUUACAGGCGUGAGCCACCGCCCCCGGCCGCCUCUGAGUUUCCAGCCUCGUUGGCCCUCCAGCCUUUUAACCUGUUGGGCCUAGGAUCAGGAAAGGUUUGUUGAAUGGGGAACUAAGAAGUGAAUUCGUUCGUUCGACAAACGUUUCCUGAGCAGCCGCUGGGUGCUAGGCGCAGUGCCAGCGCGGAAUGUCCAGGGAGACCUGGUGCCCAAAGCUUGGACCCAUCGUGAGAAAUGAGAAGCAGAUACAAAGCAGUGUGGGAGUGCAGAGGAGACAAAGCAAGCCUCAUCAGGCCCAUUGCUUGCUCUGCUCUCCCUUGUACUUACCAGUGCUUGACAAUAUACAGUUAUUUACUAGCUUGGUUAUUGACUUCCUACCCAGCACUCAGUUUUAUUCACUGCUGUAUCCUCAGUGCCUAGGACGAUGCUUGGAACGUGAUUCAUUGAUUCCAGGUAAAUCAGAGGAACAAGCAACAUGAACAGAAAUAUGUAGAAAAAGCUAUUAUGCAGAAGCAUAAUUGUUGUUUCAGAAGUCCAGCAUCUGGUGCACUUAACAAUAGAGAAUAUAUUAAACUCUUUCCAAAAUAAAAAAAAAAAAA

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Publications