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Homo sapiens (human) MIR548X host gene (MIR548XHG) URS000075DA0F_9606

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MIR548XHG: MIR548XHG is a long non-coding RNA (lncRNA) that has been reported in a limited number of studies [PMC10040790]. It is one of the genes used in the calculation of a risk score for patients, along with other lncRNAs, in the context of acute myeloid leukemia (AML) [PMC10040790]. Specifically, MIR548XHG is included in a prognostic model for AML that includes 7 risk genes and 4 protective genes [PMC10040790]. In this model, MIR548XHG is expressed at a high level in the high-risk group [PMC10040790]. Additionally, an increase in MIR548XHG expression has been observed in endometriosis [PMC10040790]. MIR548XHG has also been studied in hepatocellular carcinoma (HCC) and included as one of the nine cancer-related lncRNAs (crLncRNAs) used to construct a prognostic model for HCC patients [PMC9857215]. In this model, MIR548XHG contributes to the calculation of the risk score for each patient [PMC9857215]. Specifically, it is up-regulated and considered a risk crLncRNA when expressed at higher levels in the high-risk group [PMC9857215]. Overall, MIR548XHG has been implicated as an important gene involved in cancer prognosis and may have potential as a biomarker for AML and HCC. However, further experimental validation is needed to confirm its role and significance.

Genome locations

Gene Ontology annotations

Sequence

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CGCCACCAUUUUGGCAACCAGGAAGGGACCUCCAAAGCGGGAGAAAGCAUUUAUGGUGAGGAGAAAAGCAGCUGGUAUCACACUCUACCCAAGUCCCAAAAGCAGGUUCACAUAACUCCUGUCUUCAUUAUCUGAUAUCCGUAUGAAGAAUGUAAGAUAGAAAAAAACAUUAGCAUUUGAGAGGAUUGAAAGAGAAGCAUUCUCAGCAGAGCCAAGAUUCACUUAGAACAAAAGGUCUGCAGGUGUACCCAACAGCUCCGAAGAGACAGCGACCAUCAAGAACAGGCCAAGAUGACGAUGGCAGUUUUGUCGAAAAGAGAAGGGGGAAAUGUGGGGAAAAGCAAGAGAGAUCAGAUUGUUACUGUGUCUGUGUAGAAAGAAGUAGACAUAGGAGACUCCAUUUUCUUAUGUACUAGGAGAAAUUCUUCUGCCUUGAGAUUCUGUUAAUCUAUGACCUUACCUCCAACCCCGUGCUCUCUGAAACAUGUGCUGUGUCAACUCAGAGUUGAAUGGAUUAAGGGCAGUGCAAGAUGUGCUUUGUUAAACAGAUGCUUGAAGGCAGCAUGCUCCUUAAGAGUCAUCACCAGGACUUUCUUAUAAGCUAAUUAACAAAUUUGUACAUGGUUAACAAUUGUUUACAUUAAAUUCUAUUGGUAAAGUAAAAAAAAAAAAAAA

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Publications