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Homo sapiens (human) cancer susceptibility 20 (CASC20) URS000075ACC6_9606

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CASC20: CASC20, a long non-coding RNA, has been found to play a significant role in various biological processes, including cancer progression. Overexpression of CASC20 has been associated with the activation of RUNX2 and OSX, leading to the formation of mineralized tissue [PMC9753082]. In gastric cancer (GC), CASC20 has been shown to promote metastasis by absorbing miR-143-5p and up-regulating MEMO1. This oncogenic function of CASC20 highlights its importance in GC progression [PMC9048782]. CASC20 is a long non-coding RNA (lncRNA) that has been extensively studied in various biological contexts. In one study, it was found that CASC20 overexpression was linked to the activation of RUNX2 and OSX, resulting in the formation of mineralized tissue [PMC9753082]. This suggests that CASC20 may play a role in bone development and mineralization processes. Furthermore, CASC20 has also been implicated in gastric cancer (GC) progression. It was discovered that CASC20 promotes GC metastasis by absorbing miR-143-5p and up-regulating MEMO1 [PMC9048782]. This finding suggests that CASC20 may serve as an oncogenic factor in GC development. In conclusion, CASC20 is a lncRNA that is involved in various biological processes. Its overexpression has been associated with the activation of RUNX2 and OSX, leading to mineralized tissue formation [PMC9753082]. Additionally, it plays an important role as an oncogenic factor in GC by promoting metastasis through miR-143-5p absorption and MEMO1 up-regulation [PMC9048782]. These findings highlight the significance of further investigating the molecular mechanisms underlying the functions of CASC20.

Genome locations

Gene Ontology annotations

Sequence

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GUGCUUGGGAGACAGUGAGUACGUGAAUCUGCCUGCAGAAGUUGGGAGUGGGGGGAAUAUUAUUAGGAUUAUUAGGAAGGGCAAAGUUCAGCAUCAUCCUCUCUGCUGAAAUAACCAACAUGGUGCUCAUUAAUUGGUCAUGUGAGCACUAAGGAUAACCACCCUCCCCUCCUCAAUCAGGAUCACAAAUGCUAGCUUUCAUAUGGAUUUCAAGCUGGGUGAAAUGACUUUUUUUGCUUGUAAAGAAGAAAUGAUGAUGGAGGUCUCAUCAGGAAGUGAUGCAGAAGACUGGGACAGACGGGGGUUUCACCAUGUUGGCCAGGAUGGUCUCAAACUCCUAACCUCGCGAUCCACCCACCUCGGCCUCCCAAAGUGCUAGGAUUACAGGCGUGAGGCACUGCACCCAGCCCAUCAUAAGUAUUUUCAUGUAUCAUCAUGUAUUUUAGAAAAUGGUAUUAUUGACUAUAUAUCAGUUCAUUGUGUGCACACAUUUUAUUUAAUUAAUUUGCUCUUUUCAGUUAUUUAGUUUUCUUUUUUUGUUAUUAUAAAUUUCAUAGUAAUAUAUGUCCUUAUACAUAAAUCUGAGUAUGAAUCUAGGAUUAUUUACUUAAAAUGAAUUUCCAGACACAAAUUGUGGGGUUAAAGUGGGAGGAGCAUUUUUAAAGCUUUUGAUAUAUACCGCUAAAUUGUCUUAUAGGAAUGUAUAAAUUAUGAAACAACCAGCUGUCCAUGUGAGACCAUGCAUUUCCUUAUCCAAAUGAAGUUAAAGAAAUUAUUUGUCAGCUUUUUUGGUGGUAUGUGGUAACUCCUUUUCCUGAUUUGAAUGUUUUUCUAGUACUAGUGACUUUUUACAGGUUUUUCUUAUGUUUGUUGGUUGAGUUUUUUUUUUUAAUGUCUAUGCCUAUCUUAGUAUCUAUGCCUAUUUUAAUUUUUUAAUGUCUAUGUACUUUCUUUUGGAGCAUGUUGUAUGAUAUUUUAUAAAUAAAAGGUGUUAACUCUGUCAUUAUAGUGUAAAUAUAUUUCCCAAUAUGUUUUUAAAUUUUCAUUUAUGAUAUAUAGAAAUUUUAACUUUUAUAUAUUAAAAAUAUCUAUAUUUAAAUGACUUUGUCCAGAUGUAGUGACUCAUGCCUGUAAUCACAGAAUUUUGAGAGGCUAUGUCAAGAGGAUUGCUUGAGGCCAGGAGUUCCUGGCCAGCCUGGACGACACAGUGAGACCCCUAUCUCUAAAAAAGUUUUUUUUCAAACAAUCCUGCCACCUCAGCUUCCUAAAGUGCUGGGAUUACAGGCAUGAACCAUUGAGCCCAGGUUCUUUGUAAUAUCUUCCUCCACGUUUUUACGUAUCAAUCCUGCAUUCAAACAUUUACUCAUUCUGAAACAAAUUUUAGUAUAUGUUAUAAGGUAUUGAUCCAAAUUUUUACGUUUAUAAAUAGUUAACAUCUCCAAACCAUUUAUUGAUAUUAAUUUCACCUUCAUCAUAUUGUAAAUUAUAGUUUUGAUUUUCCUUUUUGACAAUGAAUUAUCUAAGAGGAUACUUUAGAAUUUUCAAACUUAAUAAAAAACUUUU

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Publications