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Homo sapiens (human) DSCAM antisense RNA 1 (DSCAM-AS1) URS000054C6DF_9606

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DSCAM-AS1: DSCAM-AS1 is a long non-coding RNA (lncRNA) that has been studied in various types of cancer, including thyroid cancer, endometrial cancer, pancreatic cancer, ovarian cancer, breast cancer, and tamoxifen-resistant breast cancer [PMC10028226] [PMC9313190] [PMC8973590] [PMC8156468] [PMC7482804] [PMC9798554] [PMC7353012]. However, its clinical significance and functions in these cancers are still not fully understood. In endometrial cancer, DSCAM-AS1 has been found to be positively correlated with 31 genes that are high confidence targets of miRNAs miR-373-5p, miR-371B-5p, and miR-616-5p [PMC9313190]. In pancreatic cancer, DSCAM-AS1 is notably upregulated [PMC8973590]. In ovarian and breast cancers, DSCAM-AS1 has been found to regulate the expression of QAPRT through its interaction with miR-654-3p and YBX1 respectively [PMC8156468]. Additionally, in PR-positive breast cancer cells, DSCAM-AS1 expression is reduced by progesterone treatment which further suppresses the expression of ESR1 to inhibit cell migration and invasion [PMC9798554]. Furthermore, in tamoxifen-resistant breast cancer cells derived from MCF7 cells, DSCAM-AS1 acts as a competing endogenous RNA (ceRNA) by sponging miR137 to increase the expression of EPS8 and contribute to tamoxifen resistance. Overall these studies highlight the diverse roles of DSCAM AS1 in different types of cancers.

Genome locations

Gene Ontology annotations

Sequence

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CUUUGGGAGGCUGAGGCAGGCAGAUCACCUAAGGCCAGGAAUUCGACACCAGCCUGGCCAACGUGGCAAAACCCGUCUCUACUAAAAAUACAAAAAUUAGCCGGGCGUGGUGGUGUGCGCCUGGAAUCCCAGCUACCCAGGAGGCUGAGGCAGGAGAAAUGCUGGAACCCGGGAGGCAGAGGCUGCAGUGAGCUGAGAUCAUGCCACUACUGCACUCCAGCCUGGGUGACACAGCAAGACUCCCUCUAAAAAAAGAAAAAAAGAAAAGAAAAGAAAAGAAAAUGAUAUAUCCAUGAUGAAUUAAAAUGGAGUGGAACCCACUGAUGGUAUGCAGCUGAUAAGACGCUAUAGAGAAAUGAUAUCCGGACACAUGACAAAGGAAAAGUAAACCGCUGGUUUAGAGUCUUUGGAAGGCAAUUGUAUCUGACUAGAAUUUAAUAUUAUUGUUUUUCUUUCAUUGUAUUGCAUUUAUCUGUAUGGCUAGUUAAACUGAUUUUCCAUUUAAAAGUAGUAAUGCAUUUUUUUAUAAUUACAUUGUUUUAAGUAAACAAGAAUUAUUCAAAGGACAAAAACACUAGCACAGAUGGCAUUCAAUGAUUGCAGAUAUCAUAAAGAUAGUUCAUGAAUGACAAAGAUUUGAGAAGUACUGUGUUCACGAUGCUUCCUGGAAGAGGUGGGUUAUACAGUGUUUCUUAGAGAAAGGGAUAGGGGUCUACUGCUAGGAGGGAAAGAUGAAGCUACUUAGGGGGACUAGGAAACACGCAGUUGGCCUGCCCUCUGCUUGGAGAUCACAGCCAAGGAAACACAGGAGAGGCACCCCCUGGAGGAGCUCCGUGGGCUGCAGGGCUGACGUGGCCCAGCCUGGGCAACAAGAGCGAAACCCCAUCUCAAAACCACAACAACAACAACAGGACAACAGAGAUGGACGACGGAUCGGGAAAGCCAACCAGACAGCGUGAGGCCAGGACGGAAAGAGGCACAGGGAGCUCUGCUCAGUGUCGCUACAGGGGAUCUCUCAGGCUCACAACGGGCCACUCCUCUAGGGAAGUUCUGGUCUCAUCAUGAUCCUUGUUUGGUCUCACUCCCCAUGUCCUUCUCUGUCCCUCCUCCAACUGCCAUUUAUUUAUUUAACUGAAAAAGUACCAAUCACCCACAUAGGCAUGACAUACUCAUCCAUGUACCCAUUUCUUAAAAUUGAUCAUUGUUAACAUUUGGUGUAAUUUGCUUUAUUUAUUUUUAAUGAAAUAAAUAAAACUUUACAGAAAAUGCUUUAUUUUUCUCUUUGUUCCCUCCCCAUCCUAUAUUUUUCUCCUAAAAAACCCUAUUAUCAGAAAUAUUAGUGUGUAUUCCCAGUUUCGACUUUUUAUUUUAUUACACACACACUCCCACACAUAGCUGUGACAAUGAACUUCACAUAGUAUGGUUCUGUAUUUUCUCUUUGUUUUUCAAACUUACAUAAGUAUUUUACUAUUUCUCAUGGAAAAACUCACUUUUCCCAUCCAACGUUAUGUUUCCUUAAGAUCUCUCUAUGUUGAUAUAAAGAAAUCUAGCUCAUUCUUUUUAAUGAAAAUAAAGUAUAUUUUAUGAAUGUAACUCAUGCUAACCAUGGCAAUAAAAGCUCCAUCAAGCAUGCAUUUAGU

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Publications