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Homo sapiens (human) long intergenic non-protein coding RNA 680 (LINC00680) URS00008E3957_9606

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LINC00680: LINC00680 is a long non-coding RNA (lncRNA) that has been implicated in various biological processes and diseases. It has been found to play a role in the development of arthritis by inhibiting the transcription and expression of COL2A1, a gene involved in maintaining the extracellular matrix (ECM) [PMC9710225]. Additionally, LINC00680 has been shown to stimulate the transcription and expression of MMP13, which is involved in ECM degradation [PMC9710225]. This dual effect on gene expression leads to an aggravation of ECM degradation and acceleration of arthritis development. Furthermore, knockdown experiments have demonstrated that reducing the levels of EIF4A3, LINC00680, and TTN-AS1 can significantly reduce tumor volume and improve survival rates in mice [PMC7063483]. This suggests that LINC00680 may also play a role in tumor growth. In terms of cancer research, LINC00680 has been found to be up-regulated in esophageal squamous cell carcinoma (ESCC) and other malignant tumors [PMC8900330]. This finding is consistent with RNA-sequencing results that have identified other up-regulated lncRNAs such as LUCAT1, KCNMB2-AS1, CASC15, MIAT, PTOV1-AS2, and RNF217-AS1 [PMC8900330]. Overall, these studies highlight the diverse roles that LINC00680 can play in different biological processes and diseases. Its involvement in arthritis development as well as its potential contribution to tumor growth make it an interesting target for further research.

Genome locations

Gene Ontology annotations

Sequence

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GGAGGCGGUACUUCCUCCAGCAUUUGCCGCGAGUUAUUGGCAAGUUCCCCUGCAGUUGUUUGGGCUGUCGCUGUGGCUGGUUCUGCGGUGUGCGGCCAGCCAUGGAGCGCUCUGGGCCCAGCGAAGUGACAGGCUCAGACGCAUCGGGACCGGACCCGCAGCUUGUGGUCACCAUGGGCUUCACGGGGUUCGGGGUUCGGUAAAAAAGCUCACACAUUUGACUUGGAAGCAAUGUUUGAACAAACUCAAAGAACAGCUGUGGAAAGAAGUCACAAAACACUGGGCCACCAGGAACACACCUGUAGCCCAGCAAAGUUGAGGCCAUCGACUGGCUCAUCACAACAAGGAAGGCUGUGCACCACUGGGACCCAGCAGCAGCUCAGCAGCAGCAGUGAGAGGGCCUUGGGAAAGACUUGGAGGGUUUGGCUUAUACAAGACCAGAUUACAGAGGGGUAUUGAUUUGCCUUGCCCCCGCCCCCACCAUCACAGUGUGUCCCUGUCCAGUGUUGGUGCUGUGUGGCAUUGCUUGUGUCCAUAGGAGUCCAUCAAGGCAGAGCGGCAGAGCGGCAAGCAGAGCGGCAAGGCAGGUCAGCUUCCGGCUCUUCCAGGCUGCAUGUCCCCCUCAAUAUCCUUUCAACCCAAUGGAAAUGGAUUGAAAGAGUGAAAGUCACUGGUCUCAAAGAAUCUACGGAAUUUAGCAUUUUGAAAAUCACAGAGCAAUUUUGGGUAGGUAUUCACAUGCACUAAAAAACCAGGCAAACAUCUGACUCCCAGGAUAAAGUGACUGAGAGCAGUAGAAGACUUGUUGAGCCUUAAGGAAUAAGAGGAAAAUUAUGUUAUGAAGUGCAGGAACAGCACAGACCCAGACGAGGGGAUGCAGCCCGGAGCCAGUAACCCGGCGCCUCCUCCUCCCCAGGGCGCAGCGCUGGCUGCUCCCCGGAUACCGUGAUCACACGUCCCAGGUCCUGAGAGCGCCUCAUAGGUAGACAGUUCUUUGGGACUUUUGGGACGCAGCACUUGAAGAGAGGUUUACCUAAACUUUCCUCCUGGGAGCCUGUCAUCAUUUGAAAGUAACAGAAGAAAGCUGUCAGGCUGAGGAAAGUUUCAACAGGCACUUGGUGAGAAUGGGAAGCAUCCAUUCAUUGGGAAAUCACGGGGACAGAGGAGGAAUUCACGGGAUCAGAGCACUGAGAAACUCCAGAGGCUUCUGGUUCACCGCCCUGAUUCCACAGAUGUGGGUCAGAGGCAGAGAUGGAAGUUGGAAUGGACAAGCCUGCCCUCUCCUCAGCUCUCCAUGGCUCUGUCCUCAGGCUGUUUCUGCAAUUUGACAUGAGGCCUGGCCCCAGGGAAACCAGUGGGGCCUCAUCUCCUCCCUGUGCUGCUCUGAGGUGGAGCCUUCUCAGGGUCAGUCCAGCUCUGGAGCCCCUCUCCAGCUCGGUCACCAAAGCUGUCUGGUCCCAGGGCAGUGUGGAGUGACAGGCACGCCAGCCAAUCCAAUCGGCCAUUUCAUACCCAAUGCAUGUGACUGAUAAAGAGAUAACACCCUGUCUCCAAGACACAGGCUUUACAUAAACAGCUUUAAUGACUUCUUCACUUAGGGACAAGACUCCUUAGUGGUGGAACCUCAGGCAUCCAUGCAGGAUGUAAGAAAGCUCCCGUAUAUACAUGAACUGAGAAGUGUGUAGAAGAGUAAAUACUGUAAUAGGUAAUAAAAUGUAUGUAUUUUUAUUUAUGUGCUACUUUGUCUUUCUCCCUCUCUGUGUAUAUACACAUACAUACAAAAAUGUAUUCAUUCUAUGGAAAACAUGAAAGAUGUAAUAUUCUUUACAUAUUUAAUUAAUGUAUUUUUUCUGAUCAUAUGAACACCACAAAUAUAAUUUUAUAAUUUUUUUACCUAACAAUAAGUCUUAAACAUAAAUAUACAUGGACAAUAUCAUAGUUUUUUUUUUAAUCAUAAUUAAAAUGGUUGUGAAACAAAAAAAAAAAAAA

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Publications