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Homo sapiens (human) family with sequence similarity 157 member C (FAM157C) URS00007E49F7_9606

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FAM157C: FAM157C is a long non-coding RNA (lncRNA) that has been found to be associated with various biological processes and diseases [1][PMC8262737]. FAM157C has also been found to be highly upregulated in male NCSCs after osteogenic differentiation, suggesting its involvement in bone development [2][PMC8909072]. Furthermore, the deletion of FAM157C has been observed in female patients with Turner syndrome [2][PMC8909072]. FAM157C is commonly expressed in the human bone marrow and has been identified as an autosomal regulatory hallmark of osteogenic differentiation in male adult human stem cells [2][PMC8909072]. In addition, FAM157C has been found to be upregulated in sepsis and may influence target gene expression through competitive binding to miRNA-3188 [3][PMC9707798]. It has also been reported as one of the genes associated with latent tuberculosis infection (LTBI) and non-infected latent individuals (NI-LI) [4][PMC9518923]. Furthermore, FAM157C expression has been found to be increased in PNH clone cells and positively correlated with hemolysis indicators [5][PMC9889514]. Overall, while the exact function of FAM157C is still being studied, it appears to play a role in various biological processes and diseases [1][PMC8262737].

Genome locations

Gene Ontology annotations

Sequence

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GACAUUUACAAGAACUGGCGGCGGCAGCAGCAGCAGCAGCAGCAGCAGCAGCAGCAGCAGCAGCAGCAGCAACUGGAUUUGCUUUUUCACCAGAGGAUCCAGAUUUCCCUGUGGCCUCGCAAACAAAAAAGAAGGAAGACGGAGCAGCACAGUCAUUCCUUUGUGAAAAAGGCAUUCAGAGACGGGGUUUCACCGUGUUAGCCAGGAUGGUCUCGAUCUCCUGACCUCGCGAUCCGCCCGCCUCGGCCUCCCAAAGUGCUGGGAUUACAGGCGUGAGCCACCGCGCCCGGCUUCAGCUGGUUUUUCAAAAGUCAUGGCGUGUAACUGUCCGAACAGUGAACUUCGGAGAUCCACCGGAGGGGCGAGGCCACAGCUCGACCGCGAGCACCUGAGCAUCCUGCACCGCCUGCGACGGCCGUCAGGGGGCGCGAUGCCGCCUCACAGAACCUCAAGAGGCGCCCGGGUUCCGGCACAGACGGCCUGCGGCUCCAGGGGGCGGAACCGAGCCGUCUCCUCAGGACCUACGCAGGAGGCGCCGUCAUCCCUACAGGGACACCUGAGAGGGCCCAGCCUCCUCCUCCUCAGGACCUACUCGGGAGACGCCGCUGGCUCUCUAGGAACACCUGGGGACCGUGGCCAGGGACGACCCAGCCUCCUUCUCCUCAGCUCCUCAGGAACGACUGGGGAAGCUGUGGCUUCAUGGUCCCCGAGGCUGCCCGAGGAAAGACAGAGUCUCAACCAGGCUGGAGUGCAGUGGCGUGAUCUCGGCUCACUGCAACCUCCGCCUCCCAGGAGAAUCACUUGAACUGGGAGAUGGAGGUUGCAGUGAGCUGAGAUCGCACCAGUGCACUCCAGCCUGGGCAACAGAGUGAGACUCCAUCUUAUAAAAGGAAAAAAGAAAGAAAAGAAAAAUUCCAUAUCUGAGUGUUUACUCCUGAGUUUUUGAGAUUGUUAUUAAGAU

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Publications