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Homo sapiens (human) RERE antisense RNA 1 (RERE-AS1) URS00007E47C9_9606

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RERE-AS1: RERE-AS1 is an m7G-related long non-coding RNA (lncRNA) that has been identified as a potential prognostic indicator in breast cancer (BC) [PMC9608183]. It is one of the eight m7G-related lncRNAs, along with BAIAP2-DT, COL4A2-AS1, FARP1-AS1, NDUFA6-DT, TFAP2A-AS1, LINC00115, and MIR302CHG, that were selected to construct a prognostic indicator using the LASSO Cox regression algorithm [PMC9608183]. These eight lncRNAs were also identified as diagnostic biomarkers for BC [PMC9608183]. Kaplan-Meier survival analysis showed that BAIAP2-DT, COL4A2-AS1, RERE-AS1, NDUFA6-DT, TFAP2A-AS1 and LINC00115 were associated with a good prognosis in BC [PMC9608183]. Furthermore, univariate Cox regression analysis revealed that RERE-AS1 was one of the 11 m7G-related lncRNAs tightly associated with BC prognosis [PMC9608183]. The expression levels of RERE-AS1 varied according to tumor stage and T stage in BC patients [PMC9608183]. RERE-AS1 has also been found to be elevated in patients with facioscapulohumeral muscular dystrophy (FSHD) [PMC7567883]. In this study on BC prognosis and clinicopathological characteristics, tumor immune cell infiltration and tumor mutational burden (TMB), RERE-AS1 was selected as one of the differentially expressed m7G-related lncRNAs with prognostic value in BC [PMC9608183].

Genome locations

Gene Ontology annotations

Sequence

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GCACGGAAAGAUGGCCCUGCCCUCAAGGAGCUGCAGUCUGCCCAGGAAGGCAGACAGAUAAACUGCUAUAGUUUUCCACUAUUGAGCUGAAUUUCGUAUCACCAUCAAAAGUUACCUACCUACAAGUCCCUAGUAGAGAAAAUUUAACUUCUGGCUUUAAAAAAGGGACAGAAAAGCAGGCAGAUCUGCUAGUAAGCAAACUACAGCUCCCCCGAGGAGACACACAGGCAGUGGGUAAGAGGAACAGACCCAUGGCUCCACUGAAGACACAGAGCAGCAGACGAAAAAGACAUGUCCUAAAUGUUAAAUGAAUGUGACAGAAUGAUACCAGGUGACUUCUAAGACUACAUCAUACAAAGCAACACAGCUUCUGCUGGGCUCUCCCUCUCUGGACACGUGUCUUUGAAGCCCUGAGCUGCCAGAUUAAACAGUCCGGCUUGCCUAAAGUCACCACGCUGGAGAGACCAUCAGGGAUGGAGAGGUGCUACAGAAACCCUAGCUAUUCUAGUCCCCAGCCAUUUGAGUCUUCCUAGCCCAGACUCAUAAGUGAAGAAACCUUCAAGAUGAUUCCAAUACCUCCUAUCACUUGACUGCAACCUCGUGAAAGCCUAAGCCAGACCUGCACGGUGGAGCCAUUCCUAAAUUCUGGUCCCACAGAAACUGUGAAAGAUAAUAAAUGAUCAUCAUUGCUUUA

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Publications