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Homo sapiens (human) long intergenic non-protein coding RNA 899 (LINC00899) URS000075B967_9606

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LINC00899: LINC00899 is a long non-coding RNA (lncRNA) that plays a role in various biological processes. Upon LINC00899 depletion, the intensity of EB1 staining, a marker for spindle pole and spindle, was significantly reduced in control cells [PMC7160116]. In breast cancer patients, LINC00899 expression was used to split them into two groups for survival analysis [PMC6914403]. Knockdown of LINC00899 in human bone marrow-derived mesenchymal stem cells (hBMSCs) resulted in reduced expression of osteogenesis-related genes, alkaline phosphatase (ALP) activity, and osteogenic capacity [PMC8355664]. This suggests that LINC00899 is involved in the regulation of osteogenesis-related processes. Furthermore, it was found that LINC00899 directly interacts with and inhibits the expression of miR-374a [PMC8355664]. To explore the potential mechanism by which LINC00899 exerts its regulatory functions in breast cancer, miRNAs that could interact with LINC00899 were predicted using miRcode and RNA22 software [PMC6914403]. These findings provide insights into the role of LINC00899 in spindle formation, breast cancer prognosis, osteogenesis regulation, and its interaction with miRNAs.

Genome locations

Gene Ontology annotations

Sequence

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CGGCCGCCCCCGAAGCGCUGCUGUCACCCCGGCCGCGCCCCCCAACUUUCUGCACAGUCGCGGAGCUGGAAGUUUCCGGGCUUCGCGGACACGCUGGGCUGGGUUUCAGUCGCGGCUCCGAGGUUGGCAACAAAGAGGGAAAGAAGGAGGAAAAGCAGGCCGGGGAGGGGAGGAAGAGAACCGCGCGGAGGCCGCGGCGCCGAGAGCCCCAGAACUUCCAAUUCUACCCAGAAGCUUUUUUCGUCGUGUUUUUCUCUUAGACAUGAUCCUCUCUGAGGUUGGUCCUGGGCUUCCAUACGUGAUUCAUGGAAGAGGUCUCAGCCCCAAGAGCCCCUGAGGGUACUGUCCACUCCCCCUGGAAACUUCCAGAACCUGACGUGGGGCUGAAGACAUAGAGGCUCUGAGAGUUACAUAAUUGAUUCUGACUUUGGCUGUUGGUCAACAGUGUCAUAAGGUAAAAUAAGGCUGUUGUAGAAUCUGCUCAGCCAGGGAGGGGAAAGGAGUCGGCAAUCAGGUCUCCUCCUGGGCACCUUUGUGAGGCCAGCUGGCGAGAGUGGGGGGUGACACUGAGGUCCCAGCAGCUCCAAAUGCAGGCAGAGCCCUGUCCUCAGAGAAGGUCACAGCCUAGCCAAGCCCAGCCAGGUGGAUGGGCCCACGGAACGCACAGGAACCUGGAACGGAGGUUGAAAGCAGGAAGCACAGUCUGUGACUCCCCAGCCCACUCUGCAUUCGACCACUUGGGGCCCAGAAGCUUCAGGAAAGGUGCACAAGGUCACUGGGUCCCAGUACUCCCAACAGGAAGGUCUGGUCCAGGGACAGGGCUCUUCCCGACUCCCCUUAGCCACACGCACCAGAAGUUCUGCAGUGCCCAGUGGGCAUAGCAGUCCCCAAGAAUGACCCAGCACUGAAGCUGAGCCAAAGAACUUGGGGAGCGAGCCACACCCCCUCACUCCCUCUUCGCCUGCUCCAGACUUGCCAGGCGGUUGCUUUUCGUGGGAACCGGGAUGUCCUCACCACCCUGUCCAGGGCCCAGCCCCAUGUCCCUGGCCUGCUACAGCUGGAAAAAAAAAAGAGAGAUGUUUGUUUUUAUUUGUUUAUAAAAAGAAAAGUGUUAUAUAUAUAACAUAUUAUACCUCAUGAAUACAUACAAUUAUUUGUCAAUUAACAAUAAAGAAAAAUACAGCAAGCAAAAAAGACUCUCUUCCACAAAAAUAGUGUUCAUUACAGAAAAGUACAAAAAAAAAAAAAAAAAACUAAGAGGAUAUUUAGAAUUAAGAAAAAACUAAGAGGGUAUUUAGAAUUAAAAAAUAAAAAGAAAACAAUUACCCAUGAGGUAAUUACUGAAUGCAUUUGGUUGAAAGUCCUUCUGCUAUAUUUUCCAAACUGUAUGUGUAUAUAUGUGCGCAUGCAUAUAUGUGUUUGUGUGUGUGUACACAUAUCUAUAUGAAUGGACCAUAUCGUAAGUUAUAAAUGCAUACAUAUAUUCAUGUAUAUAUAAUCAUUAGAUCAUACUAUAGGUUAUUUUACAGUCUUUUUGUUGAAUACGUUGUGAGCAUUUUAUGUCAUUAUUUUCUACAAGAUUUGAAAAAUAAAGUAUAAAUACCAGUUAAAAAAAAAAAAAAAAA

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Publications