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Homo sapiens (human) wound and keratinocyte migration associated lncRNA 2 (WAKMAR2) URS000075B835_9606

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WAKMAR2: WAKMAR2 is a gene that has been studied in various contexts [PMC8319425]. According to a correlation analysis, patients with lower WAKMAR2 expression may have a poor response to immune checkpoint inhibitors (ICIs) [PMC8319425]. In a study, it was found that overexpression of WAKMAR2 led to a decrease in the level of miR-4478, while it had no impact on the expression of miR-6068 and miR-6132 [PMC7921149]. This suggests that WAKMAR2 may have specific effects on certain microRNAs [PMC7921149]. Additionally, increased expression of WAKMAR2 and EGOT may promote viral replication in SARS-CoV-2-infected cells [PMC7999169]. This indicates that WAKMAR2 could potentially play a role in viral infections [PMC7999169]. Furthermore, it has been shown that WAKMAR2 is an important regulator in the healing of skin wounds and its deficiency may contribute to the pathogenesis of chronic wounds [PMC7921149]. This suggests that WAKMAR2 could be involved in wound healing processes [PMC7921149]. Overall, these findings highlight the potential significance of WAKMAR2 in various biological processes and its potential implications for disease outcomes [PMC8319425, PMC7921149, PMC7999169].

Genome locations

Gene Ontology annotations

Sequence

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ACUGCUUUUCUGAGAGGCCAGGUGGCAGGAUGUGGGACGACUCCAGCUGACAAAGACAGUCUAACCGUGGGGUAGGGGCUGGAGCAGGGGCCAGCGACCCACGUCUACAUGCAUACUUCUCUUACACUGCUGCUACUGGAAAAGCUGAACCCCGCGCCAGGACCCCAGCCCCCUGCAAGGACCCGUGAGCGUCUGGGAAGCUGUCUCUGGGACUGAAGCCCCCCACCUCCGCCGGGCUGGCGGCCACUGCGGUACCCUACGCCCCGUCGGGCUGGUCCUGCACAAUUUGGGAAAAAGCCGCAGCGCUUCUGCAAGGUCUACGUGGCCAUGAGCAUGCAACGCUUGGCUCCAAAAAAGACACGAAAGGAGCAAAGCGCCAACGACCACCCGAUCGGAGGGCCCGAGGGGCGCCUCUUCACCAGUCAGCUGCAGCUUAAGUUCCGUGCAUUAUCUGAAAGGAACAGCUGGCUGGAGGUAUCCAGGGCUGUCACUCCAACCUCUGCAGCAGUGACCUCAACUCCCAGCACUUCAAAACCCAGACAGAAACGUCCAACAAACUCCCAGUCCAGGAGCGCUGCAAAACCAACGCCAGUUGUUUUUCUGCAGAAAAUCAUCAACUGUGGAGAAGAAGAAGGGAAAUAAGAAAGAAAGAAAACCCUAAAAACCACCCUGGCGCCCGGGCCCGCAGGCCUCGGGCCGGCUCUGAAAAGUUUGGGCUGUGCACGUGAUGAGCGCGUAGGCGGGAGCCCCAGACAGGACCCGGGCGGGCAUUUCGAGAAAAAGCAGCGGUGACAGCCUUUGGUCCCCAUCUCCAUUGUUCCUGCCAGCUCUGGACCCCAGGCUGCAUGAGACGUAGGUCCCAGGGGACACCCGACCCCGUGGCCCCAGUCUUAGCUUCCACUGCCCCUAUCUGGCUCAUGUCUUGCUGUCUGGUGUCAUGAACUGGGAGUGCAGUAAAGAGGAGUGACAAGCCUGAGGGGCCACGUUCAUACCUGCCACUGCCAACUGUCCUGAUGUAACUGCUUUGUCAUCUUGCCUGCCAGGAUUUGUGACAAGGGCAAGAAUCUUCUGUUCCAUAUGCAACAUCUUCUGGCAGCCUUGUCCUUUUUCUGUCCUUGACGACUACAAUAACAAACAGCUGUUGCCGAGGCAUUGCUGUUGACGUGUUACCUUUGAAACCUCCCUCCUGUUAUGGAAUAAGCCUCUUCCAGAUCAUGGCUCAUUAUCAUCUAGUCUGACAAGCAGCCUUGUUGCCACGGAGACCCAAAGGGAUCAGGCGUGGCAUUUGCCUGCAUCAUCACCCCCUCCAGGGGAACUAUAAGGACUCUUCUGUGCGUCAUGCGUGGCUGUCCUGGGACUGGCUGCCACCAGACUUUUCCUGCGGGUAAAACCUAAACAAAUGAUCAGCUGCAGAUAAUAUCAAGACCUCUGUUUGAUAUGUUAAUAGUGACAGCCAGAUUUCCACAAUUAACAACGAGGUGGGAAGAAAACACUGUAGUCACCAGACUUGGGAGGAGAGGGUUUGUAUUCACAUAAACACAACCUCACGUCACUGCUUGCCACCACAAAGGGCUCUGUUCACUGUUUUGUUCUCAAAGAUCAUCCUUGCGCUCAUCCUCUGAUCUUGAAUUUCUACAUAACUUUCUCAGUUUAUAUGCCCUGUGGCAAGUGCAGCAAGCACUGUUUCCUGUUUCUAAACUUGUAGAAAAUCAUCCAUACAUCUUACAGUUGUCAGUUUUAACCAGAUAACAGUGGCACUUUGUUGCUGCUUUUUUAUCUUUAGCUUAGGUUAACAGGACCCUGGAAGUAAAGUUGUUGAUUUAUUCAAUAGAGUAUUCUCAAUUAAUUUGGCUAGAUUUCUACAUGAUUCAAAAUCUAAAAAAGUAGAAAUGCAUGCUUACAUGUCUAAGGCCUGAAAAAUUGGUAGUGACAUCCCAAAAUAAAUGAAGGUUUUAAAACAAUAAA

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Publications