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Homo sapiens (human) CBR3 antisense RNA 1 (CBR3-AS1) URS000075ACB3_9606

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CBR3-AS1: CBR3-AS1, a long non-coding RNA, was found to be highly expressed in osteosarcoma cells, particularly in the MG-63 and HOS cell lines, compared to normal human osteoblasts [PMC9092395]. The function of CBR3-AS1 in promoting invasion of lung adenocarcinoma (LAD) cells was confirmed through transwell invasion experiments [PMC7796595]. Depletion of CBR3-AS1 resulted in the inhibition of invasion in A549 and H1975 cells [PMC7796595]. Furthermore, the overexpression of β-catenin was able to rescue the invasive phenotype caused by CBR3-AS1 depletion [PMC7796595]. These findings suggest that CBR3-AS1 plays a role in promoting invasion and may be involved in the β-catenin signaling pathway [PMC7796595]. The higher expression of CBR3-AS1 in osteosarcoma cells compared to normal osteoblasts indicates its potential as a biomarker for this type of cancer [PMC9092395]. Further research is needed to elucidate the exact mechanisms by which CBR3-AS1 promotes invasion and its potential as a therapeutic target for LAD and osteosarcoma.

Genome locations

Gene Ontology annotations

Sequence

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AGCCGCGGUGUGAGGGAGCGGGAGUCUUCCUUAGCUUCUCCGCCAUGGGUGUCGCUUCGUAGCCGGGCUGCUCCGGGAAAGGCCUCGUACAGCACGCAUUCACCAGAAUCCUGUCAGCUUUCCUCUUCUCAUCCAGACGCCUGGCCAGGAUCCUCGAUAAGACCGUGACCCCCAACUUGGACACCCCAUAAGGGUCUUCUCUGUCGCCCAGGCUGGAGUGCAGUGCUGCAGUCGUGGCUCACUGUAGCGUCAGUCUCCUGAGCUCAGGAAAACUAGACAAUCCACCAGCCCAGGAGGGGACAAGCAGGCUUAUUCCUCCUCCUCGUCAUCUCUGGCUCCAGCCCCACCCUGGCCCUUGCUGGCAUUCUUCCUCUUCACGUGGCUGGGGUUGGCCACCCCAAUAGGGAAGCAGAGGGAGAAGUCAAUGUGCUUCUGGGAAUCCAGGCGGACAGUGAAGAACAGGAUAUUUACCACCUGCUCACGGACACGGAUAUGACACUGUUGAAUCAGCACAUGAGCGUGGCGGAUGGACUUAGCCAAGCCAAGCUUGAAGACCUGGGUCUGUAGGAAUCUCUAAGAAAUCAUUGAUCUUCAGGCCCAGGAUGUAAUCCAGCUUCAUCUUGCCCUCAUCCAGCACCCCAGUGCGGACCAGCCACUGCAGCAGGGCAAUGCCUUCGAACAGACGCCGUGGGUCCUUCUCAUCAAGCGUCAGCAGCUCCCGGGCGGCCUUAUGGAUCUUGACCAGGGUAAAUUUGACCCUCCAGACCUCACAUUUGUUCUGGAGCCCAUACUCACCAAUCAGCUUUAGCUUUUGGUCGAGAUGAGGUUUCUCGAAGGGUCUCUGCAGGGUCACAUAAGUUUGGCAACAAACCCAGCUCUGGGCCGCUGGCAUGUUGGCUCCACUUGCCCGUCUAUGCCUAAGCACAGGCCCUGGUCACUGAGAAAGAGCUCAAAUUUUUUAUAUAUUUCAUCUAAGGUGUUGAUUCUAUUGAUACCCUUAUCCUUUUAACAUGCCCUUAUCCUUUUAAUGUUGGUAAUGAUGCUACCUGUCAUUUCUGAGACUGGUUUGUGUCUUUCGUGUUUUUUUCUGAUCUGUCUGGCUAGAGGUUUACCAAUUGAUUUGAUUUUCCUCAAACUUCUGGUUACAAUGAUUCUCUCUCUCUCUCUUUUCCUGAUUAUAUUAAUUUCUGUUCUGAUCUUUGUUUCCUUUCUUCUGCUUACUGGAUUUUAUUUGCUCUUUUUCUGGUUUCUUACUGUAAAGGCAGAAGUCAUUGAUUUGCAUUGAUUUGAGACUUCUUUUCUAAUGUAGGCAGUAAGUGGUGUAAAUUCCCUUUUAGUAUGACUUUAGUGGCAUCUCACAUUGAUAUGUUUUCAAUUUCAUUCAGUUUAUUUUUAUUUAUUUUUUAAAUUAUUUAUUUAUUUAUUUAUUUUGAGUUGUGUAGCCCUUUAUUAGCAACUAAAAUAGAAGGUAUCUGUUGUACAACAUGGGUAGUAAUUGACUAUAACUGGAGAUUUAUUAUAUUCUAAUACAGAUCAUUUAUAAAUGCAAUUUCUUUAUUAAAAAGCUUCCACCUU

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Publications