hsa-mir-384: Hsa-mir-384 is a microRNA that has been predicted to be harbored by circMAN1A2, along with other microRNAs such as hsa-miR-494, hsa-miR-491-5p, and hsa-miR-433 [PMC5663837]. CircMAN1A2 is believed to competitively bind with hsa-mir-384 and other microRNAs, potentially affecting associated target genes [PMC5663837]. In a network analysis, TGFA was found to be targeted by hsa-mir-384 and other microRNAs mediated by circMAN1A2 [PMC5663837]. Hsa-mir-384 has been associated with the differentiation of Th17 lymphocytes in the experimental autoimmune encephalomyelitis (EAE) model [PMC8452524]. In prostate cancer cells, hsa-mir-384 was found to be differentially expressed along with other miRNAs [PMC6874298]. In colorectal cancer cells, overexpression of hsa-mir-384 was studied for its possible functions in disease progression [PMC5356701]. Hsa-mir-384 has also been investigated in breast cancer cells for its potential role in disease progression [PMC5983929]. Additionally, it has been identified as a miRNA that may have relevance in Alzheimer's disease research and therapeutic development [PMC6815273].

MIR384: MIR384 is a microRNA that has been studied in various contexts. It has been found to be upregulated in exosomes from Alzheimer's disease (AD) patients' blood, suggesting its potential as a reliable marker for AD [PMC7641266]. In colorectal cancer (CRC), MIR384 expression is downregulated and correlates with the invasiveness and migratory abilities of CRC [PMC9913554]. In poplar, MIR384 is involved in the hierarchical transcriptional regulation network of secondary cell wall (SCW) formation [PMC8633455]. It is also highly expressed in different libraries, along with other miRNAs such as miR396, miR319, and miR858 [PMC5707365]. Additionally, MIR384 has been identified as one of the downstream miRNAs involved in the regulation of SlAGO1A upon potassium stress in transgenic tomato plants [PMC8539900]. In bladder cancer cells, MIR384 mimics counteract the promotive effect of FTO overexpression on cell viability [PMC7851431]. Furthermore, MIR384 has been reported to be regulated by NEAT1 lncRNA and involved in cell growth through the STAT3 pathway [PMC9188773]. It also targets SCN3A transcript through its 3'UTR region [PMC9926024]. The transcriptional regulation of MIR384 involves binding motifs for STAT3 within its promoter region [PMC6838002]. Finally, MIR384 is involved in different pathways such as root growth modulation and cytokinin (CTK) signaling in plants [PMC7574427][PMC7574427][PMC7574427][PMC7574427], and it belongs to a family of miRNAs that contains only one member [PMC5100886][PMC5025515].